Monday, August 30, 2010

Oathtaking and Registration Instruction of New Nurse 2010

The Professional Regulation Commission reminds new nurses that they are required to bring the following when they register:

  • Duly accomplished Oath Form or Panunumpa ng Propesyonal, 
  • Current Community Tax Certificate (cedula), 
  • 2 pieces passport size picture (colored with white background and complete name tag), 
  • 1 piece 1” x 1” picture (colored with white background and complete name tag), 
  • 2 sets of metered documentary stamps, and 
  • 1 short brown envelope with name and profession;

Initial Registration Fee of P600 and Annual Registration Fee of P450 for 2010-2013 will also be collected.

Successful examinees should personally register and sign in the Roster of Registered Professionals.

The oathtaking ceremony of new nurses in Manila as well as the previous ones who have not taken their Oath of Professional will be held before the Board on Monday and Tuesday, September 20 and 21, 2010, with morning (8:00 A.M.) and afternoon (1:00 P.M.) sessions at the SMX Convention Center, SM Mall of Asia, Pasay City. All must come in their white gala uniform, nurse’s cap, white duty shoes, without earrings, hair not touching the collar and without corsage .

Oathtaking tickets for the National Capital Region (NCR) and nearby regions will be available at the Philippine Nurses Association (PNA) at 1663 F.T. Benitez Street, Malate, Manila, from September 1 - 20, 2010 on a “first come first serve ” basis.

Saturday, August 28, 2010

Top Performing Schools With Less Than 30 Examinees

Top Performing Schools With Less Than 30 Examinees

Top Performing Schools With 30 99 Examinees

Top Performing Schools With 30 99 Examinees

Top Performing Schools With 100 and More Examinees

Top Performing Schools

Top 10

1. Rayan Abogado Oliva Ateneo de Naga 86.80%
2. Aileen Ancanan Austria De Los Santos-STI College, Inc.-(Delos Santos S.N.) 86.40%
3. Allyce Joana Toledo De Leon UST 86.00%
Anna Vanessa Ang Gan UST 86.00%
4. Alyssa Leonila Dela Silva Guiam CEU-Manila 85.80%
5. Charmaine Camacho Gauiran Remedios Trinidad Romualdez Memorial School 85.60%
Vida Theresa Sibayan Gumangan Saint Louis University
Abigail Diaz Icasiano Arellano University-Manila
Weena Marie Bordeos Lim UST
John Joseph Mayo Montalbo FEU-Manila
Jan Michael Gabionza Ong Our Lady of Fatima University-Valenzuela
Joan Dioquino Tejada Remedios Trinidad Romualdez Memorial School
6. Zyrus Ronn Samson Bernasor Our Lady of Fatima University-Valenzuela 85.40%
Rouchel Anne Mañez Briones UP-Manila 85.40%
7. Peter James Bongolan Abad UP-Manila 85.20%
Merjorie May Malicay Adolfo Cebu Normal University
(Cebu State College)
Clarence Joy Lozada Custodio Saint Joseph College-Cavite City
Nico Paulo Maniago Dimal Angeles University Foundation
Kris Ray Arcelo Dumaguin Velez College
Stela Joy Ramirez Engada West Visayas State University-La Paz
Jan Christian Gomez Feliciano UST
Jose Paolo Julian Galeon Tarlac State University
(Tarlac College of Tech.)
Paulo Kristoffer Lumba Macasinag De La Salle University-Health Sciences Institute
Joy Ann Acierto Tan Notre Dame of Dadiangas College
Emer Joy Tapic Vale University of Bohol
8. Hana Kirstie San Miguel Abello UP-Manila 85.00%
Kea Tena Capio UST
Ana Francesca Caballero Centeno UST
Grace Cecile We Co UP-Manila
Michael Prince Notorio Del Rosario Lorma College
Rouella Christina Martin Fajardo UST
Joe Mari Abella Flores Cebu Normal University
(Cebu State College)
Donna May Sison Fronda Saint Joseph College-Cavite City
Renan James Sace Lim UST
Romina Tan Manaloto Our Lady of Fatima University-QC
Marian Sheryl Flores Milo Saint Louis University
Maria Kriselda Perez Rosales Lyceum of the Philippines University-Batangas,Inc
Cristina Gan Satiada Chinese General Hospital Colleg of Nursing&Liberal Arts
Laurence Lester Gamboa Tan UST
Elise Cara Kaw Teng Trinity University of Asia (Trinity-QC)
Marie Kathrina Torralba Tojong University of the Visayas-Mandaue City
Jaylyn Gabrillo Villafania Saint Louis University
9.Jamela Montoya Arcilla FEU-Manila 84.80%
Czarina Myrnelli Mamore Buenafe Northwestern University
Arcel Tiatco Cabigting Angeles University Foundation
Elaine Katrina Sigalat Cala UST
Julie Ann Del Rosario Clarin UST
Ancel Rivera De Guzman Holy Angel University
Eleanor Deloeg Dela Paz Saint Louis University
Ria Leah Oropesa Esporlas University of Perpetual Help System Dalta-Las Piñas
Elaine Medina Lapaan Saint Louis University
A Nico Nahar Idris Pajes Ateneo de Zamboanga
Ana Jeska Sana Peñaranda West Visayas State University-La Pas
Jan Roland Casinto Pomuceno Notre Dame of Dadiangas College
Angeline Villarey Rempillo Our Lady of Guadalupe Colleges
Nicael Dela Cruz Salazar Pamantasan ng Lungsod ng Pasig
Ferie Angelica Yvan Soriano Silvino FEU-Manila
Ivy Barrete Susvilla Cebu Normal University
(Cebu State College)
Kara Deneice Santos Tueres Our Lady of Fatima University-Valenzuela
Ace Brian Samaniego Verallo Our Lady of Fatima University-Valenzuela
10. Abegael Panciles Bacol Manila Doctors College 84.60%
Ramon Carlo Arpon Baring CEU-Manila
Robert Iben Barit Medical College of Northern Philippines
Livia Dedoroy Barrieses Riverside College
Maria Virginia Cinco Cuayzon Our Lady of Fatima University-Valenzuela
Kathleen Anne Palanca De Leon Chinese General Hospital College of Nursing&Liberal Arts
Mark Anthony Santos De Luna Our Lady of Fatima University-Valenzuela
Edwin Suarez Del Rosario II UST
Eunice Pablico Empeño UST
Greg Ely Cambaya Flores Our Lady of Fatima University-QC
Marcius Antonius Balcita Gacayan Union Christian College
April Joy Diane Garing Galicia Wesleyan University-Philippines-Cabanatuan City
Mary Joy Sarrosa Garbanzos University of Saint La Salle
Angeli Palisoc Garcia Trinity University of Asia (Trinity-QC)
Sara Jane Jaide Labbay Ateneo De Zamboanga
Micca Flores Lagleva UST
Razel Mae Nacua Libot Cebu Normal University
(Cebu State College)
Alexandra Basañez Macalintal Ateneo De Zamboanga
Kristine De La Cruz Macasero Cebu Normal University
(Cebu State College)
Celeste Imperial Madueño Manila Doctors College
Sallie Ria Delos Santos Malayan Lyceum of the Philippines University-Manila
Jan Paula Espiritu Martinez Universidad De Manila (City Coll. of Manila)
Kimberly Chan Mendoza Saint Louis University
Marie Paz Lacanlalay Nolasco Medical College of Northern Philippines
Robelou Lizano Ong FEU-Manila
Jodellene Fernandez Perocho CEU-Manila
Clinton Rosita Rabadon Bicol University-Polangui
Erika Genina David Ronquillo Holy Angel University
Dioliza Montenegro Sacil Universidad De Santa Isabel (Col De Sta Isabel)
Katherine Mejia Viacrusis Trinity University of Asia (Trinity-QC)

July 2010 NLE Results

Over 37,000 out of 91, 008 passed the Nurse Licensure Examination that was given last July, according to a release by the Professional Regulatory Commission.

Tuesday, August 24, 2010

Pathophysiology of Schizophrenia



The pathophysiology of schizophrenia has long remained a mystery and still today, even with various hypotheses, remains somewhat uncertain: there are too many variants; not enough consistency in findings; and, despite research, a lack of documented proof.
The most well-known and respected hypothesis with regards to the pathophysiology of schizophrenia began in the 1990s and consisted primarily of the notion there is a problem with the dopamine levels in the brain of schizophrenics.
Dopamine is both a hormone and a neurotransmitter, which means that it activates five different receptors in the brain, aptly named D1, D2, D3, D4, and D5. That said, it may not be the only neurotransmitter involved in the pathophysiology of schizophrenia. Glutamate and Serotonin have also been implicated. .
Contributing to this hypothesis is the fact that drugs administered to aid dopaminergic activity bring on schizophrenic characteristics such as psychosis, in a patient, whereas drugs administered to block them help reduce, or eliminate symptoms of schizophrenia altogether.
Additional studies affecting the pathophysiology of schizophrenia include suggestions that maternal factors such as infection, malnutrician, location of birth, season of birth, and delivery, may play a significant part in the formation and subsequent appearance of schizophrenia. Studies have shown that the worldwide rate of births affected with schizophrenia is up to 8% higher when occurring in spring or winter, though no explanation for this can be offered.


Another aspect of the pathophysiology of schizophrenia that has been explored in relative detail is that of genetics, and their relation to the likelihood of immediate relatives being born with the disease. Shockingly, it has been found that 10% of all immediate family members of an infected person will be struck down with the disease. This is specifically in relation to parents, siblings, and children. With regards to twins or other multiple births, the chances they will share the disease is 50%. Genetic reports suggest that it is the X chromosome which determines whether or not a person is infected with schizophrenia, specifically, chromosomes 1, 3, 5, and 11, however further studies are needed in order to prove this theory.
Though there are many theories and hypotheses regarding the pathophysiology of schizophrenia, there is, unfortunately, still no cure for the disease. The best a sufferer can hope for nowadays is to benefit from available medication which keeps the disease under control or in remission for the duration of time for which it is taken.

Pathophysiology of Bronchial Asthma


1. An asthma attack may occur spontaneously or in response to a trigger. Either way, the attack progresses in the following manner:
  • There is an initial release of inflammatory mediators from bronchial mast cells, epithelial cells, and macrophages, followed by activation of other inflammatory cells
  • Alteration of autonomic neural control of airway tone and epithelial integrity occur and the increased responsiveness in airways smooth muscle results in clinical manifestations (e.g. wheezing and dyspnea)
2. Three events contribute to clinical manifestations
  • Bronchial spasm
  • Inflammation and edema of the mucosa
  • Production of thick mucus, which results in increased airway resistance, premature closure of airways, hyperinflation, increased work of breathing, and impaired gas exchange
3. If not treated promptly, status asthmaticus – an acute, severe, prolonged asthma attack that is unresponsive to the usual treatment – may occur, requiring hospitalization.

Bronchial Asthma - Nursing Care Plan

Pathophysiology of Amoebiasis

When cyst is swallowed, it passes through the stomach unharmed and shows no activity while in an acidic environment. When it reaches the alkaline medium of the intestine, the metacyst begins to move within the cyst wall, which rapidly weakens and tears. The quadrinucleate amoeba emerges and divides into amebulas that are swept down into the cecum. This is the first opportunity of the organism to colonize, and its success depends on one or more metacystic trophozoites making contact with the mucosa.
Mature cyst in the large intestines leaves the host in great numbers (the host remains asymptomatic). The cyst can remain viable and infective in moist and cool environment for at least 12 days, and in water for 30 days. The cysts are resistant to levels of chlorine normally used for water purification. They are rapidly killed by purification, desiccation and temperatures below 5 and above 40 degrees.
pathophysio amebiasis
The metacystic trophozoites of their progenies reach the cecum and those that come in contact with the oral mucosa penetrate or invade the epithelium by lytic digestion.
The trophozoites burrow deeper with tendency to spread laterally or continue the lysis of cells until they reach the sub-mucosa forming flash-shape ulcers. There may be several points of penetration.
From the primary site of invasion, secondary lesions maybe produced at the lower level of the large intestine.
Progenies of the initial colonies are squeezed out to the lower portion of the bowel and thus, have the opportunity to invade and produce additional ulcers. Eventually, the whole colon may be involved.
E. histolytica has been demonstrated in practically every soft organ of the body.
Trophozoites which reach the muscularis mucosa frequently erode the lymphatics or walls of the mesenteric venules in the floor of the ulcers, and are carried to the intrahepatic portal vein.
If thrombi occur in the small branches of the portal veins, the trophozoites in thrombi cause lytic necrosis on the wall of the vessels and digest a pathway into the lobules.
The colonies increase in size and develop into abscess.
A typical liver abscess develops and consists of:
  • Central zone necrosis
  • Median zone of stoma only
  • An outer zone of normal tissue newly invaded by amoeba. Most amoebic abscess of the liver are in the right lobe.
Next to the liver, the organ which is the frequent site of extra-intestinal amoebiasis is the lungs. This commonly develops as an extension of the hepatic abscess.
Source:
Handbook of Common Communicable and Infectious Diseases 2006 Edition by Dionesia Mondejar-Navales, RN, MAEd

Pathophysiology of Liver Cirrhosis



Cirrhosis of the liver is a chronic disease that causes cell destruction and fibrosis (scarring) of hepatic tissues. Fibrosis alters normal liver structure and vasculature, impairing blood and lymph flow and resulting in hepatic insufficiency and hypertension in the portal vein. Complications include hyponatremia, water retention, bleeding esophageal varices, coagulopathy, spontaneous bacterial peritonitis, and hepatic encephalopathy.
Cirrhosis is known in three major forms. In Laennec’s (alcohol-induced) cirrhosis, fibrosis occurs mainly around central veins and portal areas. This is the most common form of cirrhosis and results from chronic alcoholism and malnutrition. Postnecrotic (micronodular) cirrhosis consist of broad bands of scar tissue and results from previous acute viral hepatitis or drug-induced massive hepatic necrosis. Biliary cirrhosis consists of scarring of bile ducts and lobes of the liver and results from chronic biliary obstruction and infection (cholangitis), and is much rarer than the preceding forms.

Pathophysiology of Congestive Heart Failure (CHF)


chf pathophysiology

Heart Failure, also known as Congestive Heart Failure, is a clinical syndrome that results from the progressive process of remodeling, in which mechanical and biochemical forces alter the size, shape, and function of the ventricle’s ability to pump enough oxygenated blood to meet the body’s metabolic requirements. Compensatory mechanisms of increased heart rate, vasoconstriction, and hypertrophy eventually fail, leading to the characteristic syndrome of heart failure: Elevated ventricular or atrial pressures, sodium and water retention, decreased cardiac output, and circulatory and pulmonary congestion. Systolic dysfunction occurs when the left ventricle is unable to relax and fill sufficiently to accommodate enough oxygenated blood returning from the pulmonary circuit. Systolic dysfunction leads to increased vascular resistance and increased afterload. Diastolic dysfunction leads to pulmonary vascular congestion.

Pathophysiology of Hypertension

pathophysio hypertension
Hypertension (high blood pressure) is a disease of vascular regulation resulting from malfunction of arterial pressure control mechanisms (central nervous system, rennin-angiotensinaldosterone system, extracellular fluid volume.) the cause is unknown, and there is no cure. The basic explanation is that blood pressure is elevated when there is increased cardiac output plus increased peripheral vascular resistance.
The two major types of hypertension are primary (essential) hypertension, in which diastrolic pressure is 90 mm Hg or higher and systolic pressure is 140 mm Hg or higher in absence of other causes of hypertension (approximately 95 % of patients); and Secondary hypertension, which results primarily from renal disease, endocrine disorders, and coarctation of the aorta. Either of these conditions may give rise to accelerated hypertension – a medical emergency – in which blood pressure elevates very rapidly to threaten one or more of the target organs: the brain, kidney, or the heart.
Hypertension is one of the most prevalent chronic diseases for which treatment is available; however, most patients with hypertension are unaware, untreated, or inadequately treated. Risk factors for hypertension are age between 30 and 70; black; overweight; sleep apnea; family history; cigarette smoking; sedentary lifestyle; and diabetes mellitus. Because hypertension presents no over symptoms, it is termed the “silent killer.” The untreated disease may progress to retinopathy, renal failure, coronary artery disease, heart failure, and stroke.
Hypertension in children is defined as the average systolic or diastolic blood pressure greater than or equal to the 95th percentile for age and sex with measurement on at lease three occasions. The incidence of hypertension in children is low, but it is increasingly being recognized in adolescents; and it may occur in neonates, infants, and young children with secondary causes.

Pathophysiology of Cholelithiasis/Cholecystitis

Cholelithiasis is the presence of stones in the gallbladder. Cholecystitis is acute or chronic inflammation of the gallbladder. Choledocholithiasis is the presence of stones in the common bile duct.
Most gallstones result from supersaturation of cholesterol in the bile, which acts as an irritant, producing inflammation in the gallbladder, and which precipitates out of bile, causing stones. Risk factors include gender (women four times as like to develop cholesterol stones as men), age (older than age 40), multiple parity, obesity, use of estrogen and cholesterol-lowering drugs, bile acid malabsorption with GI disease, genetic predisposition, rapid weight loss. Pigment stones occur when free bilirubin combines with calcium. These stones occur primarily in patients with cirrhosis, hemolysis, and biliary infections.
Acute cholecystitis is caused primarily by gallstone obstruction of the cystic duct with edema, inflammation, and bacterial invasion. It may also occur in the absence of stones, as a result of major surgical procedures, severe trauma, or burns.
Chronic cholecystitis results from repeated attacks of cholecystitis, presence of stones, or chronic irritation. The gallbladder becomes thickened, rigid, fibrotic, and functions poorly.
Complications of gallbladder disease include cholangitis; necrosis, empyema, and perforation of gallbladder; biliary fistula through duodenum; gallstone ileus; and adenocarcinoma of the gallbladder.

Pathophysiology of Leukemia

Leukemia is malignant neoplasms of the cells derived from either the myeloid or lymphoid line of the hematopoietic stem cells in the bone marrow. Proliferating abnormal and immature cells (blast) spill out into the blood and infiltrate the spleen, lymph nodes, and other tissue. Acute leukemias are characterized by rapid progression of symptoms. High numbers (greater than 50,000/mm3) of circulating blast weaken blood vessel walls, with high risk for rupture and bleeding, including intracranial hemorrhage. Lymphocytic leukemias involve immature lymphocytes and their progenitors. They arise in the bone marrows but infiltrate the spleen, lymph nodes, central nervous system (CNS), and other tissues. Myelogenous leukemias involve the pluripotent myeloid stem cells and, thus, interfere with the maturation of granulocytes, erythrocytes, and thrombocytes. Acute myelogenous leukemias (AML) and acute lymphatic leukemia (ALL) have similar presentations and courses. Approximately half of new leukemias are acute. Approximately 85 % of acute leukemias in adults are AML, and incidence of AML increases with age. ALL is the most common cancer in children, with peak incidence between ages 2 and 9.
Although the cause of leukemias is unknown, predisposing factors include genetic susceptibility, exposure to ionizing radiation or certain chemicals and toxins, some genetic disorder (Down syndromes, Fanconi’s anemia), and human T-cell leukemia-lymphoma virus. Complications include infection, leukostasis leading to hemorrhage, renal failure, tumor lysis syndrome, and disseminating intravascular coagulation.

Pathophysiology of Appendicitis

pathophysiology appendicitis
Appendicitis is inflammation of the vermiform appendix caused by an obstruction attributable to infection, structure, fecal mass, foreign body, or tumor. Appendicitis can affect either gender at any age, but is most common in males 10 to 30. Appendicitis is the most common disease requiring surgery. If left untreated, appendicitis may progress to abscess, perforation, subsequent peritonitis, and death.

Saturday, August 21, 2010

Pathophysiology of Chronic Obstructive Pulmonary Disease (COPD)

Chronic Obstructive Pulmonary Disease (COPD) is a disease characterized by airflow limitation that is not fully reversible. Airflow limitation is usually progressive and associated with an inflammatory response in the lungs stimulated by irritants. COPD includes chronic bronchitis and pulmonary emphysema. Although sometimes included in COPD, asthma is a reversible disorder and is therefore considered elsewhere.
Chronic bronchitis is chronic inflammation of the lower airways characterized by excessive secretion of mucus, hypertrophy of mucous glands, and recurring infection, progressing to narrowing and obstruction of airflow. Emphysema is the enlargement of the air spaces distal to the terminal bronchioles, with breakdown of alveolar walls and loss of elastic recoil of the lungs. The two conditions may overlap, resulting in subsequent derangement of airways dynamics (e.g., obstruction to airflow). In pulmonary emphysema, lung function progressively deteriorates for many years before the illness becomes apparent.
The most common cause of COPD is cigarette smoking. Air pollution, occupational exposures, allergens, and infections may also act as irritants. Alpha1-antitrypsin deficient is an infrequent cause. Complications include respiratory failure, pneumonia or other overwhelming respiratory infection, right heart failure (cor pulmonale), arrhythmias, and depression.

Pathophysiology of Congestive Heart Failure

Right ventricular failure occurs when the right ventricle is unable to pump blood into the pulmonary circulation. Less blood is oxygenated and pressure increases in the right atrium and systemic venous circulation, which results in edema of the extremities.
Left ventricular failure occurs when the left ventricle in unable to pump blood into systemic circulation. Pressure increases in the left atrium and pulmonary veins; then the lungs become congested with blood, causing elevated pulmonary pressure and pulmonary edema.
To compensate, the cardiac muscle hypertrophies eventually resulting in decreased ventricular compliance. Decreased compliance requires higher filling pressure to produce the same stroke volume. Increased muscle mass impedes oxygenation of the heart muscle, which leads to decreased contraction force and heart failure.
As cardiac output fails, stretch receptors and baroreceptors stimulate the sympathetic nervous system, releasing catecholamines that increase the force and rate of myocardial contraction.
This causes increased systemic resistance, increased venous return, and reduced blood flow to the limbs, viscera and kidneys.
Sweating results from sympathetic cholinergic fibers, there is extra work for the heart muscle, and there is less systemic blood flow.
The renal system responds by releasing renin-angiotensin, which sets off a chain of events – vasoconstriction, leading to increased aldosterone release, causing sodium and water retention and, in turn, increasing preload. Finally, sodium and water retention becomes excessive, resulting in signs of systemic venous congestion and fluid overload.

Pathophysiology of Myocardial Infarction

pathophysiology of Myocardial Infarction In Myocardial Infarction, inadequate coronary blood flow rapidly results in myocardial ischemia in the affected area. The location and extent of the infarct determine the effects on cardiac function. Ischemia depresses cardiac function and triggers autonomic nervous system responses that exacerbate the imbalance between myocardial oxygen supply and demand. Persistent ischemia results in tissue necrosis and scar tissue formation, with permanent loss of myocardial contractility in the affected area. Cardiogenic shock may develop because of inadequate CO from decreased myocardial contractility and pumping capacity.

Pathophysiology of Dementia

Primary Dementia
Primary dementias are degenerative disorders that are progressive, irreversible, and not due to any other condition. Specific disorders are dementia of the Alzheimer’s type (DAT) and vascular dementia (formerly multi-infarct dementia). Dementia of Alzheimer’s type demonstrates progression of symptoms from the initial stage, which is characterized by mild cognitive deficits in the area of short-term memory and accomplishment of goal-directed activity, to the final stage in which profound impairment occurs in the areas of cognition and self-care abilities. Research is ongoing.  Dementia of Alzheimer’s type believed to have multiple causative factors.

Pathoyphysiology of Pregnancy Induced Hypertension (PIH)



Preeclampsia is a characterized, by vsospasms, changes in the coagulation system, and disturbances in systems related to volume and BP control. Vasospasms results from an increased sensitivity to circulating pressors, such as angiotensin II, and possibly an imbalance between the prostaglandins prostacyclin and thromboxane A1.
Endothelial cell dysfunction, believed to result from decreased placental perfusion, may account for many changes in preeclampsia. Arteriolar vasospasm may cause endothelial damage and contribute to an increased capillary permeability. This increase edema and further decreases intravascular volume, predisposing the woman with preeclampsia to pulmonary edema.
Immunologic factors may play an important role in the development of preeclampsia. The presence of a foreign protein, the placenta, or the fetus maybe perceived by the mother’s immune system as an antigen. This may then trigger an abnormal immunologic response. This theory is supported by the increased incidence of preeclampsia or eclampsia in first-time mothers or to multiparous woman pregnant by a new partner. Preeclampsia maybe an immune complex disease in which the maternal antibody system is overwhelmed from excessive fetal antigens in the maternal circulation. This theory seems compatible with the high incidence of preeclampsia among women exposed to a large mass of trophoblastic tissue as seen in twin pregnancies or hydatidiform moles.
Genetic predisposition maybe another immunologic factor. Dekker reported a greater frequency of preeclampsia and eclampsia among daughters and granddaughters of women with a history of eclampsia, which suggests an autosomal recessive gene controlling the maternal immune response. Paternal factors are also examined.
Diets in inadequate nutrients, especially protein, calcium, sodium, magnesium, and vitamin E and C, maybe an etiologic factor in preeclampsia. Some practitioners prescribed high-protein diets (90 mg supplemental protein) without caloric restriction and moderate sodium intake in the prevention and treatment of this disorder. However, data are limited regarding the association between diet and preeclampsia.
Preeclampsia progress along a continuum from mild disease to severe preeclampsia, HELLP syndrome, or eclampsia. The pathophysiology of preeclampsia reflects alteration in the normal adaptations of pregnancy. Normal physiologic adaptations to pregnancy include increase blood plasma volume, vasodilation, and decreased systemic vascular resistance, elevated cardiac output, and decreased colloid osmotic pressure. Pathologic changes in the endothelial cells of the glomeruli are uniquely characteristic of preeclampsia, particularly in nulliparous women. The main pathogenic factor is not an increase in BP but poor perfusion as a result vasospasm. Arteriolar vasospasm diminishes the diameter of blood vessels, which impedes blood flow to all organs and raises BP. Function in organs such as the placenta, kidneys, liver and brain is deceased by as much as 40% to 60%.

Nursing Care of the Newborn

Newborns undergo profound physiologic changes at the moment of birth, as they are released from a warm, snug, dark liquid-filed environment that has met all of their basic needs, into a chilly, unbounded, brightly lit, gravity based outside world.
Within minutes after being plunged into this strange environment, a newborn’s body must initiate respirations and accommodate a circulatory system to extrauterine oxygenation.
How well the newborn makes these major adjustments depends on his or her genetic composition, the competency of the recent intrauterine environment, the care received during the labor and birth period, and the care received during the newborn or neonatal period—from birth through the first 28 days of life. (Adele Pillitteri, 2007)
Two thirds of all deaths that occur during the first year of life occur in the neonatal period. More than half occur in the first 24 hours after birth—an indication of how hazardous this time is for an infant. Close observation of a newborn for this indication of distress is essential during this period (National Center for Health Statistics, 2005).

Levels of Health Care Referral System



There are 3 different levels of health care system which are primary, secondary, and tertiary. In this post, you’ll get to know more about these health care systems. These referral systems are interlinked or interconnected to one another.

Herbal medicines

Primary Level of Care

• Devolved to cities and municipalities
• Usually the first contact between the community members and other levels of health facility.
• Center physicians, public health nurse, rural health midwives, brgy. Health workers, traditional healers.

 

Secondary Level of Care

• Given by physicians with basic health training.
• Usually given in health facilities either private owned or government operated.
• Infirmaries, municipal, district hospital, out-patient departments.

 

Tips on Making a SOAPIE

SOAPIE charting is:
S (subjective data) - chief complaint or other information the patient or family members tell you.
O (objective data) - factual, measurable data, such as observable signs and symptoms, vital signs, or test values.
A (assessment data) - conclusions based on subjective and objective data and formulated as patient problems or nursing diagnoses.
P (plan) - strategy for relieving the patient's problems, including short- and long-term actions.
I (interventions) - measures you've taken to achieve expected outcomes.
E (evaluation) - analysis of the effectiveness of your interventions.
R (revision) - changes from the original care plan"


Example:
1. Nursing Diagnosis: Nausea related to anesthetic
S: Patient states, "I feel nauseated."
O: Patient vomited 100mL of clear fluid at 2255.
A: Patient is nauseated.
P: Monitor nausea and give antiemetic as necessary.
I: Patient given Compazine 1mg IV at 2300.
E: Patient states she's no longer nauseated at 2335.


2. Nursing Diagnosis: Risk for infection related to incision sites
[notice there is no "S" charted--no subjective data to chart]
O: Incision site in front of left ear extending down and around the ear and into neck--approximately 6" in length--without dressing. No swelling or bleeding, bluish discoloration below left ear noted, sutures intact. Jackson-Pratt [JP] drain in left neck below ear with 20mL bloody drainage. Drain remains secured in place with suture.
A: No infection at present.
P: Monitor incision sites for redness, drainage, and swelling. Monitor JP drain output. Teach patient S&S [signs and symptoms] of infection prior to discharge. Monitor temperature


3. Nursing Diagnosis: Delayed surgical recovery
O: Patient oriented x 3 but groggy. Patient attempted to get OOB [out of bed] at 2245 to ambulate to bathroom but felt dizzy upon standing. Lungs sound clear bilaterally.
A: Patient is dizzy when getting OOB. Patient needs post-op education about mobility and coughing and deep-breathing exercises.
P: Allowed patient to use bedpan. Assist in getting OOB in 1 hour by dangling legs on side of bed for a few minutes before attempting to stand. Monitor blood pressure. Teach patient how to get out of bed slowly to prevent dizziness and to ask for assistance. Teach coughing and deep breathing, turning, use of antiembolism stockings.
I: Allowed patient to lie down in bed after feeling dizzy. Patient used bedpan and voided 200mL clear, yellow urine at 2245. Assisted in coughing and deep-breathing exercises and taught about turning, use of antiembolism stockings.
E: Lungs remain clear bilaterally.

How to do Focus Charting or F-DAR

Focus Charting of F-DAR is intended to make the client and client concerns and strengths the focus of care. It is a method of organizing health information in an individual’s record. Focus Charting is a systematic approach to documentation. Three columns are usually used in Focus Charting for documentation: Date and Hour, Focus, and Progress Notes. The progress notes are organized into (D) data, (A) action, and (R) response, referred to as DAR (third column).
Here is an example of a format of Focus Charting or F-DAR
Date/Hour
Focus
Progress Notes
3/7/2010 8:00pm Focus of care, this may be:
  • a nursing diagnosis
  • a sign or a symptom
  • an acute change in the condition
  • behavior
Data Action
Response
The Data Category
The data category is like the assessment phase of the nursing process. It is in this category that you would be writing your assessment cues like: vital signs, behaviors, and other observations noticed from the patient. Both subjective and objective data are recorded in the data category.

The Action Category

The action category reflects the planning and implementation phase of the nursing prosess and includes immediate and future nursing actions. It may also include any changes to the plan of care.

The Response Category

The response category reflects the evaluation phase of the nursing process and describes the client’s response to any nursing and medical care.

Examples

Pain

The focus of this problem is pain. Notice the way the D,A,R were written.
Date/Hour
Focus
Progress Notes
5/20/2010 8:00pm Pain D: >Reports of sharp pain on the abdominal incision area with a pain scale of 8 out of 10
>Facial grimacing
>Guarding behavior
>Restless and irritable
A:
>Administered Celecoxib 200mg IV
>Encouraged deep breathing exercises and relaxation techniques
>Kept patient comfortable and safe
R:
>Patient reports pain was relieved

Hyperthermia

Date/Hour
Focus
Progress Notes
5/20/2010 8:00pm Hyperthermia D: >Temperature of 38.9 OC via axilla
>Skin is flushed and warm to touch
A:
>Tepid Sponge Bath (TSB) done
7:30pm>Administered 250mg IV Paracetamol as per doctor’s order
>Encouraged adequate oral fluid intake
>Encouraged adequate rest
R:
10:00pm>Temperature decreased from 38.9 to 37.1 OC

Another Variation

This is DAR made by Jay-D Man of Slideshare.net. with some modifications made. This is a very good variation.
F1: Ineffective Breathing Pattern
D1: increase respiratory rate of 24 cpm
D2: use of accessory muscle to breath
D3: presence of nonproductive cough
F2: Hyperthermia
D1: skin warm and flush to touched
D2: increased body temperature of T= 38.9 degree celsius/axilla
F3: Fatigue
D1: less movement noted
A: 9:00am
monitored v/s and charted
regulated IVF and charted
morning care done
assessed patient needs and performed handwashing before handling the patient
advised SO to always stay on patient bedside
promote proper ventilation and a therapeutic environment
elevated the head of the bed (moderate high back rest)
provided comfort measures and provide opportunity for patient to rest
due meds given
    9:30am
    tepid sponge bath done
    instructed SO to provide blanket and let patient wear loose clothing
      F4: Discharge Plan (12:00nn)
      D1: discharged order given by Dr.Name/Time

      M – advised SO to give the ff. meds at the right time, dose, frequency and route
      E – encouraged to maintain cleanliness of the house and surroundings
      T – advised to go to follow-up consultations on the prescribed date
      H – encouraged to do chest tapping to facilitate mobilization of secretion
      O – observed for signs of super infections such as fever, black fury tongue and foul odor discharges
      D – encouraged to eat fresh vegetables and fish
      S – advised to continue praying to God and hear mass on Sunday
        2:00pm – out of the room per wheelchair with improved condition

        Conclusion

        Do you have another variation on how to do the F-DAR method? You can leave your comments below!
        References:
        Fundamentals of Nursing by Kozier and Erbs
        Image Source from here

          Anorexia Nervosa Nursing Care Plans

          Anorexia nervosa is an eating disorder that causes people to obsess about their weight and the food they eat. People with anorexia nervosa attempt to maintain a weight that’s far below normal for their age and height. To prevent weight gain or to continue losing weight, people with anorexia nervosa may starve themselves or exercise excessively.
          Anorexia nervosa is a disease that affects all organ systems, principally the cardiovascular and endocrine systems. However, complications can also involve other systems, including the GI, renal, reproductive, neurologic, orofacial, dermatologic, and hematologic symptoms.
          A typical case of anorexia nervosa involves a young person (teenager or young adult) who is mildly overweight or of normal weight and who begins a diet and exercise plan to lose weight. As she loses weight and receives initial positive reinforcement for this behavior (eg, compliments by peers on her appearance), the reward is high and causes an inability to stop this behavior once an ideal weight is achieved.
          ETIOLOGICAL THEORIES

          Psychodynamics

          The individual reflects a developmental arrest in the very early childhood years. The tasks of trust, autonomy, and separation-individuation are unfulfilled, and the individual remains in the dependent position. Ego development is retarded. Symptoms are often associated with a perceived loss of control in some aspect of life and may center on fears of sexual maturity/intimacy. Although these disorders affect women primarily, approximately 5% to 10% of those afflicted are men. Additionally, eating disorders are often associated with depression, anxiety, phobias, and cognitive problems.
          Biological

          These disorders may be caused by neuroendocrine abnormalities within the hypothalamus. Symptoms are linked to various chemical disturbances normally regulated by the hypothalamus. Furthermore, a physiological defect may make it difficult for the individual to interpret sensations of hunger and fullness.

          Family Dynamics

          Issues of control become the overriding factors in the family of the client with an eating disorder. These families often consist of a passive father, a domineering mother, and an overly dependent child. There is a high value placed on perfectionism in this family, and the child believes she or he must please others and satisfy these standards.

          NURSING PRIORITIES

          1. Reestablish adequate/appropriate nutritional intake.
          2. Correct fluid and electrolyte imbalance.
          3. Assist client to develop realistic body image/improve self-esteem.
          4. Provide support/involve SO, if available, in treatment program to client/SO.
          5. Coordinate total treatment program with other disciplines.
          6. Provide information about disease, prognosis, and treatment.

          DISCHARGE GOALS

          1. Adequate nutrition and fluid intake maintained.
          2. Maladaptive coping behaviors and stressors that precipitate anxiety recognized.
          3. Adaptive coping strategies and techniques for anxiety reduction and self-control implemented.
          4. Self-esteem increased.
          5. Disease process, prognosis, and treatment regimen understood.
          6. Plan in place to meet needs after discharge.

          Nursing diagnosis of Anorexia Nervosa and Bulimia Nervosa: NUTRITION: altered, less than body requirements may be related to inadequate food intake; self-induced vomiting and chronic/excessive laxative use possibly evidenced by body weight 15% (or more) below expected (anorexia), or may be within normal range (bulimia, binge-eating), pale conjunctiva and mucous membranes; poor skin turgor/muscle tone, edema, excessive loss of hair; increased growth of body hair (lanugo), amenorrhea, hypothermia, bradycardia, cardiac irregularities, hypotension, electrolyte imbalances.

          Desired Outcome:

          1. Verbalize understanding of nutritional needs.
          2. Establish a dietary pattern with caloric intake adequate to regain/maintain appropriate weight.
          3. Demonstrate weight gain toward expected goal range.

          Nursing intervention with rationale:

          1. Establish a minimum weight goal and daily nutritional requirements.
          Rationale: Malnutrition is a mood-altering condition leading to depression and agitation and affecting cognitive functioning/decision-making. Improved nutritional status enhances thinking ability, and psychological work can begin.
          2. Involve client with team in setting up/carrying out program of behavior modification. Provide reward for weight gain as individually determined; ignore loss.
          Rationale: Provides structured eating stimulation while allowing client some control in choices. Behavior modification may be effective only in mild cases or for short-term weight gain. Note: Combination of cognitive-behavioral approach is preferred for treating bulimia.
          3. Use a consistent approach. Sit with client while eating; present and remove food without persuasion and/or comment. Promote pleasant environment and record intake.
          Rationale: Client detects urgency and reacts to pressure. Any comment that might be seen as coercion provides focus on food. When staff member responds consistently, client can begin to trust her or his responses. The single area in which client has exercised power and control is food/eating, and she or he may experience guilt or rebellion if forced to eat. Structuring meals and decreasing discussions about food will decrease power struggles with client and avoid manipulative games.
          4. Provide smaller meals and supplemental snacks, as appropriate.
          Rationale: Gastric dilation may occur if refeeding is too rapid following a period of starvation dieting. Note: Client may feel bloated for 3–6 weeks while body readjusts to food intake.
          5. Make selective menu available and allow client to control choices, as much as possible.
          Rationale: Client who gains self-confidence and feels in control of environment is more likely to eat preferred foods.
          6. Be alert to choices of low-calorie foods/beverages; hoarding food; disposing of food in various places such as pockets or wastebaskets.
          Rationale: Client will try to avoid taking in what is viewed as excessive calories and may go to great lengths to avoid eating.
          7. Maintain a regular weighing schedule, such as Monday/Friday before breakfast in same attire, on same scale, and graph results.
          Rationale: Provides accurate ongoing record of weight loss/gain. Also diminishes obsessing about changes in weight.
          8. Weigh with back to scale (depending on program protocols).
          Rationale: Although some programs prefer client to see the results of weighing, this approach can force the issue of trust in client who usually does not trust others.
          9. Consult with dietitian/nutritional therapy team.
          Rationale: Helpful in determining individual dietary needs and appropriate sources. Note: Insufficient calorie and protein intake can lower resistance to infection and cause constipation, hallucinations, and liver damage.
          10. Transfer to acute medical setting for nutritional therapy, when condition is life-threatening.
          Rationale: The underlying problem cannot be cured without improved nutritional status. Hospitalization provides a controlled environment in which food intake, vomiting/elimination, medications, and activities can be monitored. It also separates the client from SO(s) and provides exposure to others with the same problem, creating an atmosphere for sharing.
          Nursing Diagnoses

          1. Imbalanced Nutrition: Less Than Body Requirements

          Common Related Factors Defining Characteristics
          Severe fear of obesity Severely distorted self-concept, self-esteem, and/or body image
          Absence of physical conditions that would explain weight loss or prevent weight gain
          Body weight 15% to 29% or more below ideal weight for height Self-restricted calorie intake despite hunger
          Obsession with food, calories, weight, and control issues
          Common Expected Outcomes Patient stops losing weight.
          Patient begins to gain weight.
          Patient recognizes eating disorder.
          NOC Outcomes Nutritional Status: Food and Fluid Intake; Weight Control
          NIC Interventions
          Eating Disorders Management; Weight Gain Assistance; Nutritional Therapy

          Ongoing Assessment

          Actions/Interventions Rationale
          Record the patient’s weight and height on intake. Weigh regularly, maintaining standard conditions (i.e., same scale, same time of day, patient wearing similar clothes). This ensures accurate record of weight changes.
          Weigh the patient in a matter-of-fact manner without discussion. This reduces risk of acting-out behaviors. Weight gain is only one aspect of the total therapeutic program; other critical factors include nutritional adequacy, behaviors related to eating, appropriate use of exercise, and development of a healthy body image.
          Obtain weight history, including initial motivation for weight loss or food restrictions. Clinical anorexia can follow ordinary weight loss dieting.
          Conduct a nutritional assessment: It is critical that the health care provider openly discuss and have an understanding of the complex food and weight-related behaviors of the patient so that appropriate supports can be integrated into the treatment plan.
          • Assess the patient’s beliefs and fears about food and weight gain
          Excessive focus on food and weight can be a maladaptive method of coping with stress.
          • Knowledge about nutrition and sources of information
          This information provides the basis for an individualized teaching plan about maintaining adequate nutritional intake.
          • Behaviors used to reduce calorie intake (dieting), to increase energy output (exercising), and generally to lose weight (vomiting, purging, and laxative abuse)
          This provides data on patient thinking and thought distortions.
          Assess cardiovascular, metabolic, renal, gastric, hematological, and endocrine system functioning. Assessment provides data on the severity of malnutrition.
          Monitor intake (i.e., daily food plans that track eating trends along with emotional states and triggering events). Record intake and output for the hospitalized patient. These data help determine the patient’s actual caloric intake and eating behaviors.

          Therapeutic Interventions

          Actions/Interventions Rationale
          Prescribe appropriate nutrition and total calories per day to relieve acute starvation. A gradual refeeding prescription ensures steady weight gain and reduces risk of medical complications.
          Supervise all activities immediately before and after meals; maintain supervision consistency. This decreases opportunity to engage in compensatory activities to reduce calorie intake.
          Provide food and meals without comment. This helps separate emotional behaviors from eating behaviors.
          Set limits on all exercise but allow daily activity. Preventing all forms of exercise may induce severe anxiety.
          Assure the patient that treatment is not designed to produce obesity. Patients have an overwhelming fear of weight gain and obesity.
          Acknowledge any anger, sadness, or feeling of loss that the patient may have toward treatment. This helps provide external emotional controls that have not yet been internalized by the patient.
          Provide supplemental feedings and nutrition as indicated. Nutritional supplements may be necessary if the patient is malnourished. Tube or parenteral feedings may be necessary if the patient is unable to allow herself or himself oral feedings.

          2. Disturbed Body Image

          Common Related Factors Defining Characteristics
          Difficulty coping with development and maturation Inability to achieve unreasonable personal goals
          Alexithymia (channeling uncomfortable feelings into behaviors such as self-starvation)
          Distorted views of one’s body weight and shape for age Negative feelings about self and body
          Self-loathing (impulsive or obsessive)
          Intense fear of gaining or not being able to lose weight
          Common Expected Outcomes Patient identifies positive thoughts and feelings regarding body and self.
          Patient identifies a direct means of coping with problems.
          NOC Outcome Body Image
          NIC Interventions
          Self-Awareness Enhancement; Body Image Enhancement

          Ongoing Assessment

          Actions/Interventions
          Rationale
          Explore the patient’s understanding of his or her physical body, especially as it relates to maturation. Assess to what degree the patient’s negative body image and negative self-concept are related to overwhelming anxiety. Patients with anorexia have a distorted body image.
          Assess to what degree culture, religion, race, and gender influence the patient’s negative views of self. Cultural and social norms about body size and shape may influence the patient’s thinking and feelings about his or her body image.
          Determine the family or patient’s perceptions regarding psychological and physical changes brought about by anorexia. These data need to be compared to the patient’s thinking prior to the onset of anorexia.
          Obtain the patient’s assessment of personal strengths and weaknesses. Patients learn they have the ability to handle day-to-day stress.
          Assess the patient’s ability to identify “here and now” emotional states and precipitating events that trigger negative behaviors. The patient may not be aware of the relationship between feelings and eating behaviors.

          Therapeutic Interventions

          Actions/Interventions
          Rationale
          Encourage reexamination of positive and negative self-perceptions. The patient needs to develop a realistic understanding of his or her body image.
          Encourage the patient to identify the differences between “real people” and celebrities. Patients often use media reports of celebrities as a guide for their eating behaviors.
          Encourage recognition, expression, and acceptance of unpleasant feelings. Patients with anorexia have a need for control in multiple areas of their lives. Mastery over food may have become a method for reducing tensions.
          Help the patient develop a realistic, acceptable perception of body image and food. Patients must understand the complex health problems associated with anorexia.
          Refer the patient to individual counseling and a support group for eating disorders. Multiple approaches are needed to achieve long-term changes in behavior. Groups that come together for mutual support and guidance can provide long-term assistance.

          Acute Pain Nursing Diagnosis

          Acute Pain NCP

          2 Diabetic Retinopathy Nursing Care Plans

          Diabetic retinopathy is retinopathy (damage to the retina) caused by complications of diabetes mellitus, which can eventually lead to blindness. It is an ocular manifestation of systemic disease which affects up to 80% of all patients who have had diabetes for 10 years or more. Despite these intimidating statistics, research indicates that at least 90% of these new cases could be reduced if there was proper and vigilant treatment and monitoring of the eyes.

          Altered Sensory Perception
          In poorly controlled DM, microcirculatory changes occur rapidly wherein the lining of the retinal blood vessels that supply the retina is blocked within the retina. Visual impairment occurs as a result of this    ischemia which significantly alter the visual perception of the patient.
          Assessment Nursing Diagnosis Objectives Nursing Interventions Rationale Expected Outcome
          s> the pt. may be verbalize: -change in sensory acuity
          o>
          -lethargy
          -dependency on significant others
          -bilateral blindness
          -change in usual response to stimuli
          -poor concentration
          - change in behavior pattern
          Altered sensory perception (visual) r/t micro vascular destruction 2° DM Short term: after 1-2 hrs. of NI, the pt. will demonstrate behaviors to prevent accidents that can result due to visual impartment. Long term:
          After 2-3 days of NI. The pt. will be free of injury caused by the sensory alterarion.
          >establish rapport >monitor and assess V.S
          >assess pt’s general physical condition
          >listen and respect clients expression of dependency
          >reorient to time, place, and staff and events as necessary
          >explain procedures / expected sensations and outcomes
          >minimize discussion of negatives within clients hearing
          >provide rest periods
          >arrange bed, personal articles and food trays
          >instruct SO not to leave the client unattended
          >provide safety measures
          >position door s and furniture’s so they are out of travel path
          >administer antidiabetic medications as needed
          >to gain pt’s. trust and cooperation >to obtain baseline data
          >to note for any abnormality
          >to encourage verbalization of feelings
          >enhances pt’s. well being and sense of importance.
          >to promote participation
          >pt may misinterpret and believe references are to himself / herself
          >to conserve energy
          >enhances safety
          >to prevent injury
          >to aid in maintaining balance
          >to treat underlying condition.
          The pt shall have demonstrated behaviors to prevent accident / injures. The pt shall have been free of injury caused by the alteration
          Risk For Falls
          Retina is the most metabolically active structure per weight of tissue in the body. Thus the retina is an inevitable target for micro vascular damage in diabetes mellitus. Diabetic retinopathy appears to be a response to retinal ischemia resulting from blood vessel changes and RBC aggregation. It is the leading cause of blindness. In the case of the pt. retinopathy become a chronic complication that lead to blindness. Blindness increase the pt’s susceptibly to falling that may cause physical harm. This impartment in eyesight may to inability to recognize danger.
          Assessment Nursing Diagnosis Objectives Nursing Interventions Rationale Expected Outcome
          s> the patient may verbalize: -weakness
          -imbalance of stance and posture
          o>
          -elderly
          -needs  assistance in performing ADLs
          -dependent to significant others
          -decrease Hgb concentration
          -bilateral blindness
          -(+) homans sign on both legs
          - with  limited ROM
          -faintness when turning
          -decrease lower extremely strength
          -difficulty c̄ gait
          Risk for falls r/t eyesight impairment (blindness) Short term: After 1-2 hrs of NI, the pt will be able to demonstrate behaviors to reduce and protect  self from injury.
          Long term:
          After 2-3 days of NI, the pt will be free of injury from mechanical sources
          >establish rapport >monitor and assess V.S
          >assess pts general physical condition
          >evaluate client’s cognitive status
          >provide information regarding clients dse  condition (s) that may result in increase risk of falls.
          >review medication regimen and how it affects clients. Instruct in monitoring of side effect.
          >ensure safety precautions
          >instruct the S.O not to leave the unattended .
          >instruct pt. to ask for assistance during ambulation/ transfer
          >administer due to meds
          >to gain pts trust and cooperation >to obtain  baseline
          >to note any abnormality
          >affects ability to perceive own limitation and risk for falling
          >to instill awareness on the need to promote safety.
          >use of pain medication may contribute to weakness and confusion; multiple medications and combinations  may contribute to dizziness or loss of balance
          > to prevent falls
          >to treat underlying medical condition
          The pt shall have demonstrated behaviors to protect self from injury The pt shall have been free for injury.

          CVA Nursing Care Plan

          CVA Nursing Care Plan

          Aplastic Anemia Nursing Care Plan

          Aplastic anemia is a condition where bone marrow does not produce sufficient new cells to replenish blood cells. The condition, per its name, involves both aplasia and anemia. Typically, anemia refers to low red blood cell counts, but aplastic anemia patients have lower counts of all three blood cell types: red blood cells, white blood cells, and platelets, termed pancytopenia.
          Signs and Symptoms:
          • Anemia with malaise, pallor and associated symptoms such as palpitations
          • Thrombocytopenia (low platelet counts), leading to increased risk of hemorrhage, bruising and petechiae
          • Leukopenia (low white blood cell count), leading to increased risk of infection
          • Reticulocytopenia (low reticulocyte counts)
          Causes
          In many cases, the etiology is considered to be idiopathic (cannot be determined), but one known cause is an autoimmune disorder in which white blood cells attack the bone marrow.

          Risk for Infection

          Aplastic anemia is characterized by the depression of hemato-poietic activity in bone marrow affecting all blood cells.  In effect, serum level of RBC, WBC and Platelets are depleted.  Apparently, WBCs or leukocytes are the ones responsible in acting against foreign microorganisms that invade the body. Such event of a depleted leukocytes can highly predisposes the individual to acquire various secondary infec-tions since the body is said to be immuno-compromised.  Thus, a nursing diagnosis of risk for infection was derived.
          Assessment Nursing Dx Planning Nursing Inter­ventions Rationale Expected Outcome
          S = O O = the client manifested the ff. s/sx:
          • weakness
          • easy fatigability
          • dryness of lips
          • bloodstained gums and teeth
          • with untrimmed and dirty fingernails
          • with limited range of motion
          • lab values suggest a decreased in WBC count, neutrophils, hematocrit, hemoglobin and
          • thrombocytes
          = the client may further manifest:
          -hyperthermia
          -flushed skin
          -irritability
          -restlessness
          -various signs of different systemic infection such as:
          Resp: Cough, colds, adventitious breath sounds
          Skin: rashes, itchiness, wounds, untimely wound healing
          GI: nausea, vomiting, abdominal discomfort
          GU: dysuria, abnormal discharges and other associated symptoms
          Risk for Infection r/t inadequate secondary defenses 2 Aplastic Anemia Short term: After 4 hours of NI, the client will identify interven-tions to reduce/ prevent risk for infection.
          Long term:
          After 3 days of NI, the client will display techniques and lifestyle changes conducive for her health promotion and disease prevention and she will be free from signs of potential infection.
          1.       Maintain established rapport 2.       Monitor vital signs, especially temp.
          3.       Stress proper hand washing techniques by all caregivers between therapies
          4.       Monitor visitors/ caregivers of the client.  Instruct SO to limit visitors especially those with known contagious illnesses.
          5.       Maintain aseptic/sterile techniques as much as possible
          6.Advise the use of facial mask when going outside the client’s room
          7.       Encouraged early ambulation, deep breathing and turning exercises as indicated.
          8. Provide regular catheter/ perineal care as indicated
          9.       Reinforce teachings about diet.  Avoid raw meats, fruits and vegetables.  Consume prescribed nutritionally adequate menus.
          10.    Enforce strict bed rest.  Provide the client her favorite books as necessary.
          11. Review to the client and SO about the nature of the disease and the interventions needed.
          12.    Instruct the client to report significant changes that she may experience typical to a presence of infection.
          13. Refer to other members of the health care team.
          1. to gain trust & relieve anxiety 2.  to obtain baseline data; also, fever is usually the initial sign of infection
          3.       hand washing is the primary measure against nosocomial infections/ cross contamination
          4.       prevents unwanted exposure of the client to other communicable diseases
          5.  to avoid cross contamination
          6. reverse isolation is an important for clients who are immuno-suppressed
          7. simple exercises helps tone body built and strengthen the body system.
          8.  to prevent growth  of microorganisms
          9.  to provide an optimum nutrition to meet daily needs of client; raw food can contain microorganisms that may precipitate infection
          10.    client is in great need of staying on her room to avoid potential exposure to pathogenic elements
          11.  helps alleviate anxiety; can also foster cooperation and compliance to prescribed therapeutics
          12. to intervene with such events accordingly
          13.    promotes inter-disciplinary care rendered to the client
          Short term: The client shall have identified interventions to reduce/prevent risk for infection
          Long term:
          The client shall have displayed techniques and lifestyle changes conducive for her health promotion and disease prevention and have been free form signs of potential infection.